Cleveland BioLabs, Inc. (NASDAQ:CBLI) Files An 8-K Results of Operations and Financial Condition

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Cleveland BioLabs, Inc. (NASDAQ:CBLI) Files An 8-K Results of Operations and Financial Condition
Item 2.02 Results of Operations and Financial Conditions.

On November 14, 2017, Cleveland BioLabs, Inc. (the “Company”) issued a press release announcing its financial results for the quarter ended September 30, 2017. The information in this Item 2.02 of Form 8-K and Exhibit 99.1 attached hereto shall not be deemed “filed” for purposes of Section 18 of the Securities Exchange Act of 1934, as amended, or otherwise subject to the liabilities under that section, nor shall it be deemed incorporated by reference in any filing under the Securities Act of 1933, as amended, except as shall be expressly set forth by specific reference.

Item 9.01 Financial Statements and Exhibits.

(d)

Exhibit


CLEVELAND BIOLABS INC Exhibit
EX-99.1 2 a2017q3pressrelease.htm EXHIBIT 99.1 Exhibit Exhibit 99.1FOR IMMEDIATE RELEASECLEVELAND BIOLABS REPORTS THIRD QUARTER 2017 FINANCIAL RESULTS AND DEVELOPMENT PROGRESSBuffalo,…
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About Cleveland BioLabs, Inc. (NASDAQ:CBLI)

Cleveland BioLabs, Inc. (CBLI) is a biopharmaceutical company. The Company’s programs are focused developing approaches to activate the immune system and address serious medical needs. Its platform of Toll-like immune receptor activators has applications in mitigation of radiation injury and immuno-oncology. Its advanced product candidate is entolimod, an immuno-stimulatory agent, which the Company is developing as a radiation countermeasure and an immunotherapy for oncology and other indications. It also has an additional clinical-stage program and multiple projects in different stages of preclinical drug development. Its products include Entolimod, CBLB612, CBL0137 and Mobilan. Entolimod is a toll-like receptor 5 (TLR5). CBLB612 is a compound-based upon a natural activator of another tissue-specific component of the immune system. Mobilan is a recombinant non-replicating adenovirus that directs expression of TLR5.

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