Esperion Therapeutics,Inc. (NASDAQ:ESPR) Files An 8-K Other EventsItem 8.01Other Events
On May23, 2018, Esperion Therapeutics,Inc. issued a press release titled, “Esperion Announces Third Pivotal Phase 3 Study of Bempedoic Acid Meets Primary Endpoint” (the “Press Release”). A copy of the Press Release is filed herewith as Exhibit99.1 and is incorporated herein by reference.
Item 8.01Financial Statements and Exhibits.
(d)Exhibits
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ExhibitNo.
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Description
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99.1
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Press Release dated May23, 2018.
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Esperion Therapeutics, Inc. ExhibitEX-99.1 2 a18-14203_1ex99d1.htm EX-99.1 Exhibit 99.1 Media Contact: Elliot Fox W2O Group 212.257.6724 efox@w2ogroup.com Investor Contact: Alex Schwartz Esperion 734-249-3386 aschwartz@esperion.com Esperion Announces Third Pivotal Phase 3 Study of Bempedoic Acid Meets Primary Endpoint Study 3 Achieves Additional 26% LDL-C Lowering on Background of Maximally Tolerated LDL-C Lowering Therapy in Patients Considered Statin Intolerant Additional hsCRP Reduction of 25% Cumulative Phase 2 / Phase 3 Demonstrates Broad Efficacy as well as Safety and Tolerability Conference Call and Webcast on Wednesday,…To view the full exhibit click here
About Esperion Therapeutics,Inc. (NASDAQ:ESPR)
Esperion Therapeutics, Inc. is a pharmaceutical company. The Company is focused on developing and commercializing oral, low-density lipoprotein cholesterol (LDL-C) lowering therapies for the treatment of patients with elevated LDL-C. The Company’s segment is the business of researching, developing and commercializing therapies for the treatment of patients with elevated LDL-C. The Company’s lead product candidate is ETC-1002, or bempedoic acid. The Company is engaged in conducting a global Phase III long-term safety and tolerability study of bempedoic acid in patients with hyperlipidemia whose LDL-C is not adequately controlled with low- and moderate-dose statins. Bempedoic acid is an inhibitor of ATP Citrate Lyase (ACL), a well-characterized enzyme on the cholesterol biosynthesis pathway. Bempedoic acid inhibits cholesterol synthesis in the liver, decreases intracellular cholesterol and up-regulates LDL-receptors.